Bartonellosis: A Serious, yet Ignored Disease

Bartonellosis

This disease is no joke. But since the media and corrupt bureaucracies can’t make a profit off of the testing and treatment, mainstream doctors overlook the disease and rarely diagnose it. Bartonellosis can lead to mild to severe (even fatal) infections if they aren’t promptly treated. Transmission can occur from dogs, cats, other animals. In addition, fleas, spiders, bed bugs, ticks, horseflies, and many other vector borne insects can transmit the disease. Many mainstream studies today speak of Bartonella as an “emerging disease”. However, in the sources below, there is evidence of Bartonella being a problem in the 1920’s.

Given the severity of the disease and how easily it’s spread by several hosts; why aren’t people freaking out about Bartonella, like they are any of the other media driven diseases? Bartonella is heavily linked to Autoimmune Disease, Neuroimmune Diseases (i.e Fibromyalgia, Myalgic Encephalomyelitis, Gulf War Illness), and can cause severe neurologic and psychiatric manifestations in those who contract the disease.

Also, many doctors and leading health agencies (who dont forget, only release information on diseases that make them profits or benefit the status quo) claim that Bartonellosis is only a problem in immunocompromised patients. This is a problem for two different reasons. First, it doesn’t take into account the type of immune suppression that is completely denied by the medical establishment (i.e. Lyme/antibody deficiency, vax injury, etc.)

Second, Bartonella itself, causes immune suppression; which allows it to spread and also lead to reactivated viruses and opportunistic infections (see in soruces below).

https://globalscience.home.blog/2019/11/30/anatomy-of-a-cover-up/

To make matters worse, like any of the disease covered up by the CDC; testing isn’t very accurate. Even private labs that do specialized tests, can miss cases of Bartonellosis. Sadly, very few doctors will treat this infection without positive serological evidence. Even though its written in literature that it’s a clinical diagnosis.

Even with a positive Bartonella test, most mainstream medical workers will not properly treat the infection (most likely because the big brothers at the CDC will take their practicing liscence away). Most people will have to pay out of pocket for their doctors and treatment for Bartonellosis. In countries like Canada, people are forced to pay for private doctors or leave the country (which they can no longer do), if they wish to heal from this devastating infection.

In my own personal battle with Bartonella, i almost died. It destroyed my body, caused unimaginable pain, caused neurologic hell, and affected my mental health in brutal fashion. Its an unimaginable beast. It took me three years, loads of money, pharmaceuticals, herbs, and a complete lifestyle change to put Bartonellosis remission. And in order to maintain remission, it requires very strict life style decisions, balancing essential vitamins and nutrients, and periodic treatment with herbs. Its no joke. I hope this post can help bring proper awareness to this debilitating and multifaceted disease.

Worst of all, people who keep pushing the government and media narrative are helping enforce strict policies that are preventing children, women, and men world wide from accessing doctors who can treat infections like Bartonellosis.

Here is a ton of information on Bartonellosis:

  1. “Bartonella spp. are responsible for emerging and re-emerging diseases around the world. The majority of human infections are caused by Bartonella henselae, Bartonella quintana and Bartonella bacilliformis, although other Bartonella spp. have also been associated with clinical manifestations in humans. The severity of Bartonella infection correlates with the patient’s immune status. Clinical manifestations can range from benign and self-limited to severe and life-threatening disease. Clinical conditions associated with Bartonella spp. include local lymphadenopathy, bacteraemia, endocarditis, and tissue colonisation resulting in bacillary angiomatosis and peliosis hepatis. Without treatment, Bartonella infection can cause high mortality. To date, no single treatment is effective for all Bartonella-associated diseases. In the absence of systematic reviews, treatment decisions for Bartonella infections are based on case reports that test a limited number of patients. Antibiotics do not significantly affect the cure rate in patients with Bartonella lymphadenopathy. Patients with Bartonella spp. bacteraemia should be treated with gentamicin and doxycycline, but chloramphenicol has been proposed for the treatment of B. bacilliformis bacteraemia. Gentamicin in combination with doxycycline is considered the best treatment regimen for endocarditis, and erythromycin is the first-line antibiotic therapy for the treatment of angioproliferative lesions. Rifampicin or streptomycin can be used to treat verruga peruana. In this review, we present recent data and recommendations related to the treatment of Bartonella infections based on the pathogenicity of Bartonella spp.”

https://www.sciencedirect.com/science/article/abs/pii/S0924857914001186

  1. “Ocular complications associated with Bartonella henselae infection may include central retinal artery and vein occlusion, neovascular glaucoma, and severe vision loss.”

https://www.sciencedirect.com/science/article/abs/pii/S0002939403007840

  1. “A remarkable feature of the genus Bartonella is the ability of a single species to cause either acute or chronic infection and either vascular proliferative or suppurative manifestations. The pathological response to infection with Bartonella spp. varies substantially with the status of the host immune system. Indeed, infection with the same Bartonella species (eg, B. henselae) can result in a focal suppurative reaction (CSD in immunocompetent patients), a multifocal angioproliferative response (BA in immunocompromised patients), endovascular multiplication of the bacteria (endocarditis), or an exaggerated inffammatory response without evidence of bacteria in patient tissues (meningoencephalitis)(86).”

https://aac.asm.org/content/48/6/1921.short

  1. “Five species of Bartonella have been found to infect humans, henselae, quintana, elizabethae, bacilliformis, and vinsonii. The most common of these in North America are Bartonella quintana and Bartonella henselae, the agents of trench fever, bacillary angiomatosis, and parenchymal peliosis, and in the case of B. henselae cat-scratch disease. B. bacilliformisis endemic in Peru and Ecuador, where it causes oroya fever or Carrion’s disease. New methods of diagnosing Bartonella infections have resulted in increased recognition of the many manifestations of these infections. Early recognition is crucial, as these are potentially fatal opportunistic infections that usually respond rapidly to appropriate antimicrobial therapy.”

https://www.annualreviews.org/doi/abs/10.1146/annurev.med.47.1.355

  1. “Clinical presentation of infection with Bartonella ranges from a relatively mild lymphadenopathy with few other symptoms, seen in cat scratch disease, to life-threatening systemic disease in the immunocompromised patient. In some individuals, infection manifests as lesions that exhibit proliferation of endothelial cells and neovascularization, a pathogenic process unique to this genus of bacteria. As the spectrum of disease attributed to Bartonella is further defined, the need for reliable laboratory methods to diagnose infections caused by these unique organisms also increases. A brief summary of the clinical presentations associated with Bartonella infections is presented, and the current status of laboratory diagnosis and identification of these organisms is reviewed”

https://cmr.asm.org/content/10/2/203.short

  1. “There are now at least 13 named species and probably an equal number of unnamed species. Of these, only four have been definitively associated with human disease: B. henselae, B. quintana, B. bacilliformis, and B. elizabethae. B. quintana infection may be asymptomatic or may be characterized by high fever, severe shin pain, and relapsing symptoms over weeks to months. The species most commonly associated with endocarditis in humans is B. quintana, and most patients with B. endocarditis require replacement of the infected cardiac valve. Granulomatous inflammatory disease (CSD), most commonly of the lymph nodes, occurs in immunocompetent humans infected with B. henselae. In humans and cats, B. quintana and B. henselae bacteremia, respectively, persist despite the development of a humoral antibody response”

http://www.buhnerhealinglyme.com/category/co-infections/bartonella/

  1. Bartonella henselae can infect humans resulting in a wide range of disease syndromes including cat-scratch disease, fever with bacteremia, endocarditis, bacillary angiomatosis, and bacillary peliosis hepatis, among others. The nature and severity of the clinical presentation correlates well with the status of the hosts’ immune system. Individuals with impaired immune function, including HIV infection, progress to systemic infections more often. Patients with intact immune function who become infected with B. henselae usually get cat-scratch disease, a disease that usually involves lymphadenopathy resulting from a strong cellular immune response to the bacterium. However, immunocompromised patients often progress to bacillary angiomatosis or bacillary peliosis hepatis. The reduced ability of the hosts immune response to control bacterial infection apparently results in a bacteremia of longer duration, and in some patients the presence of angiogenic lesions that are unique among bacterial infections to Bartonella. Recently, the role of immune effector cells that produce angiogenic cytokines upon stimulation with B. henselae has been proposed. Here, the current status of the role of the immune response in both controlling infection and in B. henselae-triggered immunopathogenesis is presented”

https://www.liebertpub.com/doi/abs/10.1089/104454903767650694

  1. “Bartonella spp. are small gram-negative rods, which use to live as intracellular parasites in endothelial cells and erythrocytes of human and animal hosts. Bartonella quintana infections were common in Europe during World War I, when it caused epidemics of “trench fever”(Maurin and Raoult 1996). Bartonella endocarditis was not reported at that time.”

https://link.springer.com/article/10.1007%2Fs12223-013-0235-9

  1. “Bartonella spp are fastidious bacteria that occur in the blood of man and mammals; they are usually vector borne but can also be transmitted by animal scratches and bites. The bartonelloses of medical importance comprise Carrión’s disease, trench fever, cat-scratch disease, bacillary angiomatosis, and peliosis hepatis. Carrión’s disease, known as Oroya fever in the acute phase and verruga peruana (Peruvian wart) in its chronic form, has curious manifestations that, until recently, have been restricted in their geographic distribution to dwellers of the high, dry Andean valleys, but new sites of disease are emerging. Trench fever is associated with louse-borne disease and homelessness. Cat-scratch disease, bacillary angiomatosis, and peliosis hepatis are increasingly being recognized as causes of human disease, especially in susceptible population groups such as HIV-infected persons. The Bartonella spp are considered emerging human pathogens. The clinical manifestations, differential diagnosis, laboratory diagnosis, and treatment of these conditions are discussed”

https://www.sciencedirect.com/science/article/abs/pii/S0738081X08002186

  1. Bartonella resembles AIDS. Causing reactivated viruses and opportunistic infections.

“Human bartonellosis is a South American anthroponosis caused by Bartonella bacilliformis. The disease has an acute phase characterized by invasion of red blood cells by parasites, and consequent severe anemia; and a chronic phase presenting with benign vascular tumors. During the acute phase, affected individuals are prone to developing opportunistic infections with a variety of organisms similar to the ones seen in AIDS. After antibiotic treatment is instituted, a subgroup of patients may develop atypical symptoms which potentially represent clinical manifestations of the restoration of macrophage function. We speculate that the pathophysiology of the acute phase of human bartonellosis resembles AIDS, with a period of immunosuppression following the infection and later, clinical manifestations of immune reconstitution subsequent to treatment.”

https://www.sciencedirect.com/science/article/abs/pii/S0306987709004708

  1. “CSD may manifest with various ocular pathological findings. Taking into consideration the increasing frequency of infections by B. henselae and B. quintana, clinicians should always incorporate CSD in the differential diagnosis of such presentations of uveitis. Educating vulnerable groups (children, immunosuppressed, etc.) and also general population, the appropriate preventing measures can contribute in limiting the risk of infection.”

https://link.springer.com/article/10.1007/s10792-019-01096-7http://www.gordonmedical.com/unravelling-complex-chronic-illness/bartonella-like-organisms-blo-consideration-signs-and-symptoms/

  1. They’ve known about Bartonella species since 1926:

“The intradermal injection of the culture into the eyebrow of young macaques gives rise to nodular formations rich in new blood vessels and showing the bacilliform organism within the endothelial cells. From the experimentally induced nodules cultures of the organism are readily recovered.”

https://rupress.org/jem/article/43/6/851/9509/ETIOLOGY-OF-OROYA-FEVER-I-CULTIVATION-OF

  1. Another one from 1927:

“Bartonella bacilliformis has been detected microscopically, though in small numbers, in all tissues showing histological changes. Parallel cultural determinations of the presence of Bartonella bacilliformis in the blood, liver, spleen, lymphatic glands, bone marrow, and local lesions established the relationship between the pathological conditions and the presence of the parasite. The organism seems to persist longest in the lymphatic glands. Cultural methods offer a simple and conclusive means for the determination of the presence or absence of the infecting organism.”

https://rupress.org/jem/article-standard/45/3/437/9598/ETIOLOGY-OF-OROYA-FEVER-VI-PATHOLOGICAL-CHANGES

  1. An ignored etiology in mental illness?

“Three patients presented with acute psychiatric disorders associated with Bartonella-like signs and symptoms. Each had clear exposure to ticks or fleas and presented with physical symptoms consistent with Bartonella, eg, an enlarged lymph node near an Ixodes tick bite and bacillary angiomatosis found only in Bartonella infections. Laboratory findings and the overall general course of the illnesses seemed consistent with Bartonella infection. The authors are not reporting that these patients offer certain proof of Bartonella infection, but we hope to raise the possibility that patients infected with Bartonella can have a variety of mental health symptoms. Since Bartonella can clearly cause neurologic disorders, we feel the presence of psychiatric disorders is a reasonable expectation.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2100128/

  1. A Groundhog, a Novel Bartonella Sequence, and My Father’s Death

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3044516/

16.”In this preregistered case-controls study performed on 250 healthy subjects tested earlier for the presence of anti-Toxoplasma IgG antibodies, we searched for the positive association between presence of anamnestic anti-Bartonella IgG antibodies and depressiveness measured with Beck II inventory, depression subscale of neuroticism measured with N-70 questionnaire, and self-reported health problems. We found that that Bartonella seropositivity was positively correlated with Beck depression only in Toxoplasma-seronegative men and negatively correlated with health in Toxoplasma-seronegative women. Bartonella seropositivity expressed protective effects against Toxoplasma seropositivity-associated increased neuroticism in men while Toxoplasma-seropositivity expressed protective effects against Bartonella seropositivity-associated health problems in women. A comparison of the patterns of association of mental and physical health problems with Bartonella seropositivity and with reported cat-related injury suggests that different factor, possibly infection with different pathogen transmitted by cat related-injuries than the B. henselae, is responsible for the observed association of cat related-injuries with depressiveness and major depression. “
https://www.frontiersin.org/articles/10.3389/fpsyt.2018.00314/full

  1. “Bartonella spp. are facultative intracellular pathogens that employ a unique stealth infection strategy comprising immune evasion and modulation, intimate interaction with nucleated cells, and intraerythrocytic persistence. Infections with Bartonella are ubiquitous among mammals, and many species can infect humans either as their natural host or incidentally as zoonotic pathogens. Upon inoculation into a naive host, the bartonellae first colonize a primary niche that is widely accepted to involve the manipulation of nucleated host cells, e.g., in the microvasculature. Consistently, in vitro research showed that Bartonella harbors an ample arsenal of virulence factors to modulate the response of such cells, gain entrance, and establish an intracellular niche. Subsequently, the bacteria are seeded into the bloodstream where they invade erythrocytes and give rise to a typically asymptomatic intraerythrocytic bacteremia. While this course of infection is characteristic for natural hosts, zoonotic infections or the infection of immunocompromised patients may alter the path of Bartonella and result in considerable morbidity”

https://cmr.asm.org/content/25/1/42.short

  1. “Bartonella spp. and Brucella spp. are closely related α-proteobacterial pathogens that by distinct stealth-attack strategies cause chronic infections in mammals including humans. Human infections manifest by a broad spectrum of clinical symptoms, ranging from mild to fatal disease. Both pathogens establish intracellular replication niches and subvert diverse pathways of the host’s immune system. Several virulence factors allow them to adhere to, invade, proliferate, and persist within various host-cell types. In particular, type IV secretion systems (T4SS) represent essential virulence factors that transfer effector proteins tailored to recruit host components and modulate cellular processes to the benefit of the bacterial intruders. This article puts the remarkable features of these two pathogens into perspective, highlighting the mechanisms they use to hijack signaling and trafficking pathways of the host as the basis for their stealthy infection strategies.”

http://m.perspectivesinmedicine.cshlp.org/content/3/8/a010231.short

  1. “Bartonella species are emerging human pathogens responsible for a wide range of clinical manifestations, including Carrion’s disease, trench fever, cat-scratch disease, bacillary angiomatosis-peliosis, endocarditis and bacteraemia. During infection of their human or animal reservoir host(s), these arthropod-borne pathogens typically invade and persistently colonize mature erythrocytes. However, in both reservoir and incidentally infected hosts, endothelial cells are target cells for bartonellae. Endothelial interactions involve a unique mode of cellular invasion, the activation of a proinflammatory phenotype and the formation of vasoproliferative tumours. Based on the establishment of bacterial genetics and appropriate infection models, recent work has begun to elucidate the cell and molecular biology of these unusual pathogen–host cell interactions.”

https://www.sciencedirect.com/science/article/abs/pii/S0966842X01020479

  1. The CDC doing what they do best and avoiding conducting tests on ticks being able to transmit Bartonella spp.

“Case studies and serological or molecular surveys involving humans, cats and canines provide indirect evidence supporting transmission of Bartonella species by ticks. Of potential clinical relevance, many studies have proposed co‐transmission of Bartonella with other known tick‐borne pathogens. Currently, critically important experimental transmission studies have not been performed for Bartonella transmission by many potential arthropod vectors, including ticks.”

https://onlinelibrary.wiley.com/doi/full/10.1111/j.1365-2915.2008.00713.x

  1. Proof that ticks transmit Bartonella spp:

“The potential role of ticks as vectors of Bartonella species has recently been suggested. In this study, we investigated the presence of Bartonella species in 271 ticks removed from humans in Belluno Province, Italy. By using primers derived from the 60-kDa heat shock protein gene sequences, Bartonella DNA was amplified and sequenced from four Ixodes ricinus ticks (1.48%). To confirm this finding, we performed amplification and partial sequencing of the pap31 protein and the cell division protein FtsZ encoding genes. This process allowed us to definitively identify B. henselae (genotype Houston-1) DNA in the four ticks. Detection of B. henselae in these ticks might represent a highly sensitive form of xenodiagnosis. B. henselae is the first human-infecting Bartonella identified from Ixodes ricinus, a common European tick and the vector of various tickborne pathogens. The role of ticks in the transmission of bartonellosis should be further investigated”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2958531/

  1. Transmission:

“Among the many mammals infected with Bartonella spp., pets represent a large reservoir for human infection because most Bartonella spp. infecting them are zoonotic. Cats are the main reservoir for Bartonella henselae, B. clarridgeiae, and B. koehlerae. Dogs can be infected with B. vinsonii subsp. berkhoffii, B. henselae, B. clarridgeiae, B. washoensis, B. elizabethae, and B. quintana. The role of dogs as an important reservoir of Bartonella spp. is less clear than for cats because domestic dogs are more likely to be accidental hosts, at least in nontropical regions. Nevertheless, dogs are excellent sentinels for human infections because a similar disease spectrum develops in dogs. Transmission of B. henselae by cat fleas is better understood, although new potential vectors (ticks and biting flies) have been identified. We review current knowledge on the etiologic agents, clinical features, and epidemiologic characteristics of these emerging zoonoses.”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3291446/

  1. Bartonella in dog saliva

“Bartonella species, transmitted by arthropods or animal bites and scratches, are emerging pathogens in human and veterinary medicine. PCR and DNA sequencing were used to test oral swabs collected from dogs. Results indicated the presence of 4 Bartonella species: B. bovis, B. henselae, B. quintana, and B. vinsonii subspecies berkhoffii.”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2876763/

  1. “Bartonella also known as cat-scratch fever is a bacterial infection. Symptoms include swollen, painful lymph nodes, muscle and/or joint pain, nausea, vomiting, chills, anxiety, insomnia, red rashes that resemble stretch marks. Treatment is often Levaquin or Rifampin, Biaxin or Azithromycin. Septra/Bactrim has shown in some cases to be effective.”
  1. “It is well known that co-infections may occur with Lyme Disease relatively frequently. Patients with a history of Lyme Disease who have incomplete resolution of symptoms should be evaluated for Bartonella infections. Bartonella is an intracellular, gram-negative bacteria that can become chronic. Certain lab tests may not detect the infection due to a variety of strains and the lack of sensitivity of the tests. It is advised to use both PCR and IFA methods of testing and not to dismiss the disease due to negative tests when symptoms are present. Various Bartonella species have been recognized since the early 1950s.
    Bartonella may not present in its usual form when additional infections, such as Lyme or Babesia are present. In addition, typical Bartonella lesions are not always seen in patients, therefore, a diagnosis of “fever of unknown origin” should alert a physician to consider Bartonella. It is estimated that approximately 2/3 of the patients with Bartonella have a fever. Involvement of practically every organ has been reported.”

http://www.lymeinfo.net/bartonella.html

26.”The study identified a much larger tick-borne disease (TBD) burden within the Australian community than hitherto reported. In particular, the first cases of endemic human Babesia and Bartonella disease in Australia with coexisting Borrelia infection are described, thus defining current hidden and unrecognized components of TBD and demonstrating local acquisition in patients who have never been abroad.”
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3258014/

  1. “Ticks are the vectors of many zoonotic diseases in the United States, including Lyme disease, human monocytic and granulocytic ehrlichioses, and Rocky Mountain spotted fever. Most knownBartonella species are arthropod borne. Therefore, it is important to determine if some Bartonella species, which are emerging pathogens, could be carried or transmitted by ticks. In this study, adult Ixodes pacificus ticks were collected by flagging vegetation in three sites in Santa Clara County, Calif. PCR-restriction fragment length polymorphism and partial sequencing of 273 bp of the gltA gene were applied forBartonella identification. Twenty-nine (19.2%) of 151 individually tested ticks were PCR positive for Bartonella. Male ticks were more likely to be infected with Bartonellathan female ticks (26 versus 12%, P = 0.05). None of the nine ticks collected at Baird Ranch was PCR positive forBartonella. However, 7 (50%) of 14 ticks from Red Fern Ranch and 22 (17%) of 128 ticks from the Windy Hill Open Space Reserve were infected with Bartonella. In these infected ticks, molecular analysis showed a variety of Bartonella strains, which were closely related to a cattle Bartonella strain and to several known human-pathogenic Bartonella species and subspecies: Bartonella henselae, B. quintana, B. washoensis, and B. vinsonii subsp.berkhoffii. These findings indicate that I. pacificus ticks may play an important role inBartonella transmission among animals and humans.”

http://jcm.asm.org/content/39/4/1221.short

28.”Bartonella quintana, the aetiologic agent of trench fever, has recently been implicated in culture-negative endocarditis and bacteraemia amongst homeless people. B. quintana is a fastidious slow-growing organism. A tissue culture system of human endothelial cells was developed in which B. quintana grew intracellularly. Observation of the different steps during infection of these cells demonstrated that the bacteria adhered to and penetrated the cells by phagocytosis. During the preadherence stage, most bacteria exhibited surface appendages that resembled those described for Salmonella typhimurium and which may mediate specific interactions between the eucaryotic cell and the bacterium. Soon after the engulfment step, the bacterium appeared in a cell vacuole where it multiplied, giving the typical aspect of morulae which has also been reported with Ehrlichiae or Chlamydiae. In older cultures, the coexistence of bacteria and huge quantities of vesicle-like structures in the same vacuole were noted. These vesicle-like structures were also found with agar-grown bacteria and were identified as membrane blebs. Microscopic observation of heart valves from B. quintana endocarditis patients demonstrated the intracellular location of B. quintana in vivo. This intracellular location of B. quintana should now be considered in further studies on the pathogenesis of the diseases it causes.”

http://www.sciencedirect.com/science/article/pii/S0923250897851194

  1. “PCR analysis of Ixodes scapularis ticks collected in New Jersey identified infections with Borrelia burgdorferi (33.6%), Babesia microti (8.4%), Anaplasma phagocytophila (1.9%), and Bartonella spp. (34.5%). The I. scapularis tick is a potential pathogen vector that can cause coinfection and contribute to the variety of clinical responses noted in some tick-borne disease patients.”
    http://jcm.asm.org/content/42/6/2799.short

30.”Our data implicate B henselae as a potential human tick-borne pathogen. Patients with a history of neuroborreliosis who have incomplete resolution of symptoms should be evaluated for B henselae infection.”
http://archneur.jamanetwork.com/article.aspx?articleid=780359

31.CDC talking about the severity of Bartonella.

“Bartonella spp. infection has been reported in association with an expanding spectrum of symptoms and lesions. Among 296 patients examined by a rheumatologist, prevalence of antibodies against Bartonella henselae, B. koehlerae, or B. vinsonii subsp. berkhoffii (185 [62%]) and Bartonella spp. bacteremia (122 [41.1%]) was high. Conditions diagnosed before referral included Lyme disease (46.6%), arthralgia/arthritis (20.6%), chronic fatigue (19.6%), and fibromyalgia (6.1%). B. henselae bacteremia was significantly associated with prior referral to a neurologist, most often for blurred vision, subcortical neurologic deficits, or numbness in the extremities, whereas B. koehlerae bacteremia was associated with examination by an infectious disease physician. This cross-sectional study cannot establish a causal link between Bartonella spp. infection and the high frequency of neurologic symptoms, myalgia, joint pain, or progressive arthropathy in this population; however, the contribution of Bartonella spp. infection, if any, to these symptoms should be systematically investigated.”
http://wwwnc.cdc.gov/eid/article/18/5/11-1366_article?commit=GO

  1. Lyme Disease and Bartonella: More Common Than You Think
  1. “To investigate the role of B. henselae in patients with symptoms suggesting neuroborreliosis, serum and cerebrospinal fluid samples were tested with serological and PCR methods. Among 17 examined patients, in 12 cases Borrelia burgdorferi infections were detected, in 1 case Bartonella henselae infection was ascertained, and in two patients mixed B. burgdorferi and B. henselae infections were found. These results indicate that mixed infections should be taken into consideration in establishing diagnosis of neurological disorders. Further study of this conclusion is needed.”
  1. “Various friends of the author have been afflicted with Lyme disease and/or bartonellosis. Their suffering has motivated him to gather the following information from doctors, online articles, afflicted friends, and personal experience in watching a loved one suffer. The accuracy of the technical information is dependent upon the accuracy of what he has heard and read from medical professionals and online articles including Wikipedia (although Wikipedia can be edited by anyone, it is heavily monitored and moderated so that the vast majority of information is provided by learned authors).”

https://www.benbrew.com/lb/lb.html

  1. “Bartonella henselae is a hitherto unidentified cause of autoimmune haemolytic anaemia. Here we report a case of Coombs‐negative autoimmune haemolytic anaemia. The episode was preceded by exposure to a cat and a non‐specific infectious syndrome. Concomitant serum titres of B. henselae antibodies were indicative of a recent infection. The case report suggests that B. henselae infection can trigger secondary autoimmune haemolytic anaemia.”

https://onlinelibrary.wiley.com/doi/full/10.1046/j.1365-2141.2001.03165.x

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